Magnetic resonance imaging of the wrist and hand

Bones

The wrist is perhaps the most complex joint in the body and is affected by a wide spectrum of disorders. It is composed of the distal radius and ulna, carpal bones, and five metacarpal bases. Eight carpal bones articulate to form the carpus, which can be divided into two horizontal rows (Figure 6) [26]. The proximal carpal row is composed of the scaphoid, lunate, and triquetrum. The pisiform is considered a sesamoid bone and not part of the proximal carpal row [30]. However, it can indirectly affect the triquetrum. The distal carpal row is composed of the trapezium, trapezoid, capitate, and hamate. These bones form an arch that supports the metacarpals and is considered more stable than the proximal row.

The carpal vascular anatomy is considered atypical because the blood vessels often enter the bones distally. This pattern of blood supply places the proximal segments of the bones at risk for osteonecrosis or nonunion after fracture. A single vessel supplies the scaphoid, capitate, and the lunate in approximately 20% of people, which places these bones most at risk for developing osteonecrosis [30]. The trapezoid and hamate have two non-articular nutrient arteries but lack consistent intraosseous anastomosis, which also places fracture fragments at risk for osteonecrosis. The trapezium, triquetrum, and pisiform bones in approximately 80% of people demonstrate redundant blood supply making osteonecrosis in these bones rare [30].

Ulnar variance refers to the relative lengths of the distal radial and ulnar articular surfaces. It can be classified as positive (ulnar surface longer), neutral (both articular surfaces of the same length), or negative (ulnar surface shorter). The ulnar styloid process can be variable in length and is independent of ulnar variance. Wrist position can alter the degree of ulnar variance. As a result, although ulnar variance can be suggested on MRI, using this modality to diagnose and measure the degree of variance is inappropriate. Therefore, determination of subtle ulnar variance on MRI should be done with caution [31].

Triangular fibrocartilage complex

The TFCC lies on the ulnar side of the wrist and acts as the primary stabiliser of the distal radioulnar joint (DRUJ). It is composed of the triangular fibrocartilage disc (TFC), the dorsal and volar radioulnar ligaments, the ulnar collateral ligament (UCL, between the ulnar styloid process and triquetrum), the meniscus homologue, and the extensor carpi ulnaris (ECU) tendon sheath (Figure 10) [26,32]. The TFC is thicker peripherally than centrally, giving it a biconcave appearance on sagittal images. It attaches to the distal margin of the sigmoid notch at the ulnar aspect of the distal radius over the articular cartilage. The peripheral attachment of the TFC to the distal ulna is named the triangular ligament and has a proximal and a distal attachment, known as the proximal and distal laminae. The proximal lamina attaches to the distal ulnar fovea at the base of the ulnar styloid. The distal lamina attachment is variable and extends to and beyond the ulnar styloid process; it blends with the fibrous connective tissue of the ECU tendon sheath. The space between the proximal and distal laminae of the triangular ligament is termed the ligamentum subcruentum, which represents loose, vascularised tissue. Increased signal within the ligamentum subcruentum on PD images can be normal and should not be misinterpreted as a tear [33]. The meniscus homologue (ulnomeniscal homologue) is composed of fibrous tissue between the ulna and triquetrum. It has a common origin with the dorsal radioulnar ligament and inserts on the ulnar aspect of the triquetrum and lunate [26,32].

Figure 10

Illustrated diagram of the triangular fibrocartilage complex

Intrinsic ligaments

The wrist consists of many ligaments, classified as intrinsic (interosseous) and extrinsic (capsular). The intrinsic ligaments connect and are interposed between the carpal bones, and are important for maintaining the integrity of the proximal carpal row. The extrinsic ligaments connect the carpal bones either to the radius and ulna or the metacarpal bones. Injuries may result in partial-thickness, full-thickness incomplete, and full-thickness complete tears.

The most important ligaments for supporting the proximal carpal row, and those that are most commonly injured, are the scapholunate (SLL) and lunotriquetral (LTL) ligaments. The SLL is C-shaped and consists of dorsal, central, or membranous, and volar or palmar bands. The dorsal and volar bands are true ligaments; on the other hand, the central component is composed of fibrocartilage. The dorsal band of the SLL is the most important for stability of the wrist [34] and is homogenously low in signal intensity on MRI (Figure 11). The other two bands show intermediate signal resulting from the presence of fibrovascular components. The membranous components of both the SLL and LTL are best depicted on coronal MRA and computed tomography arthrographic images.

Figure 11

Normal scapholunate (SLL) and lunotriquetral (LTL) ligament anatomy. A) Axial T2W fat-suppressed (FS) magnetic resonance (MR) image through the level of the carpus demonstrate the scaphoid (S), lunate (L), and triquetrum (T). The dorsal bands of the SLL (long arrow) and LTL (curved arrow) are seen. The volar bands of the SLL (short arrow) and LTL (arrowhead) are also noted. B) Coronal T2W FS MR image demonstrates the normal triangular shaped appearance of the central component of the SLL (white arrow) and LTL (arrowhead)

The LTL is V-shaped and also made of three components: dorsal, central, or membranous, and volar or palmar bands. The LTL is smaller than the SLL. The volar component blends with the TFCC and is the strongest. However, the dorsal component is the most important in carpal stability.

Extrinsic ligaments

The extrinsic wrist ligaments are divided into palmar/volar and dorsal structures (Figure 12). The volar extrinsic wrist ligaments are stronger than the dorsal ligaments and more important for proper wrist function, but show variable anatomy (Figure 12A). The most important ones are the volar radioscaphocapitate ligament, which prevents rotatory subluxation of the scaphoid and the radiolunatotriquetral ligament (also known as the long radiolunate ligament), which connects the distal radius with the proximal carpal row (Figure 12A and C). The radioscaphocapitate ligament originates from the distal aspect of the radial styloid and extends ulnarly and distally to attach to the capitate. The long radiolunate ligament originates from the radial styloid, adjacent to the origin of the radioscaphocapitate ligament, and extends obliquely to attach on the lunate. The deep fibres of this ligament blend with the LTL and attach to the triquetrum [34]. The additional volar extrinsic ligaments are the short radiolunate, radioscapholunate and palmar scaphotriquetral. Two ulnar-sided volar extrinsic wrist ligaments, the volar ulnolunate and volar ulnotriquetral ligaments, originate from the volar radioulnar ligament and insert on the lunate and triquetral bones, respectively (Figure 10). These ligaments contribute to the stability of the TFCC [26,34].

Figure 12

Normal extrinsic wrist ligaments. A) Illustration of the major volar extrinsic ligaments. B) Illustration of the major dorsal wrist extrinsic ligaments and collateral ligaments. C) Coronal 3D T1W fat-suppressed gradient echo post arthrogram magnetic resonance image shows portions of the volar radioscaphocapitate (arrows) and long radiolunate (arrowhead) ligaments

The most important dorsal extrinsic ligament is the dorsal radiocarpal or radiolunatotriquetral ligament. An additional dorsal capsular wrist ligament is the dorsal intercarpal ligament which extends from the scaphoid to the dorsal tubercle of the triquetrum and variably to the second metacarpal base (Figure 12B) [26,34]. Some authors describe an additional dorsal ulnotriquetral ligament.

The radial collateral ligament (RCL) of the wrist is located between the radial styloid and scaphoid, and the UCL is located between the ulnar styloid and triquetrum. These ligaments do not play a significant role in wrist stability (Figure 12B).

Extrinsic wrist ligaments can be depicted with MRI and MRA, including 3D isotropic imaging, and have a striated appearance. All imaging planes are used for their assessment.

Carpal tunnel, including median nerve and flexor tendons

The carpal tunnel is an anatomic space overlying the volar aspect of the carpal bones (Figure 13A). Its volar surface is the flexor retinaculum, which extends from the distal radius to the metacarpal bases. The flexor retinaculum can be divided into three components, including the antebrachial fascia, transverse carpal ligament, and distal aponeurosis between the thenar and hypothenar muscles. The ulnar margin consists of the hook of the hamate, triquetrum, and pisiform bones. The radial margin of the carpal tunnel consists of the scaphoid, trapezium, and the flexor carpi radialis tendon sheath [26,35].

Figure 13

Illustrated diagrams depicting the wrist in cross-section. A) Illustration of the wrist flexor and extensor tendons in the distal wrist at the level of the carpal tunnel. B) Illustration of the wrist flexor and extensor tendons in the wrist at the distal radioulnar level

The carpal tunnel contains nine tendons including the four flexor digitorum superficialis (FDS) tendons, four flexor digitorum profundus (FDP) tendons, and the flexor pollicis longus tendon. The FDS and FDP tendons are located in the ulnar bursa, while the flexor pollicis longus tendon is located within the radial bursa. The ulnar bursa is most frequently in continuity with the fifth digit flexor tendon sheath, while the second to fourth digits usually have separate flexor tendon sheaths. The radial bursa is in continuity with the flexor pollicis longus tendon sheath at the volar aspect of the thumb [36]. There is anatomic variability of the bursae, and the two bursae may communicate with one another or the ulnar bursa may occasionally be continuous with the second to fourth finger flexor tendon sheaths [37]. The flexor carpi radialis tendon has its own osteofibrous tunnel, and is separated from the carpal tunnel by the deep portion of the transverse carpal ligament, which splits along its radial border, surrounding this tendon and bridging across the trapezium.

The palmaris longus tendon is present in 86% of the population and is located superficially to the flexor retinaculum [38]. It inserts onto the central part of the flexor retinaculum and lower part of the palmar aponeurosis. This muscle assists with wrist flexion; however, it is considered functionally insignificant. Its greatest importance can be to serve as a donor for tendon grafting [39].

The median nerve is the most superficial structure at the radial aspect of the carpal tunnel between the flexor retinaculum and flexor tendons of the index and middle fingers. On MRI, the median nerve has a rounded or slightly flattened appearance with intermediate signal intensity and a relatively constant calibre from DRUJ up to the level of the hook of the hamate.

The flexor tendons are situated at the volar aspect of the wrist and extend distally with the majority attaching onto the metacarpal bones and phalanges. The origins and insertions of the wrist and hand muscles and tendons are described in Table 1.

Extensor tendons

There are six extensor tendon compartments along the dorsum of the wrist, deep to the extensor retinaculum, which extend distally to attach onto the metacarpal bones and phalanges (Figure 13B). The origins and insertions of the wrist and hand muscles and tendons are described in Table 2.

Common anatomic variants

There are numerous normal osseous, muscular, and nerve anatomic variants that can either mimic pathology or be a cause of symptoms. Listing each described variation is beyond the scope of this article. However, the most common and clinically relevant variants are described here.

Within the bones there are variant morphologies of some of the carpal bones, including developmental coalitions and numerous reported developmental ossicles. For instance, a lunotriquetral coalition is the most common carpal coalition, with a prevalence of 0.1%, and it represents a congenital fusion of the lunate and triquetral bones [41]. Osseous coalitions of the lunate and triquetrum are typically asymptomatic. However, fibrocartilaginous LTL coalitions can be associated with atypical ulnar-sided wrist pain.

Two lunate morphologies have been described. A type I lunate has a single distal facet with the capitate and does not articulate with the hamate, whereas a type II lunate has two distal facets with the radial facet articulating with the capitate and ulnar facet articulating with the hamate. This can be associated with focal lunohamate osteoarthritis and ulnar-sided wrist pain.

There are numerous accessory ossicles of the wrist that can be mistaken for pathology or occasionally be a source of symptoms. Two of the most commonly encountered ossicles are the os styloideum and the os hamuli. The os styloideum is a dorsal accessory ossicle between the second and third metacarpal bases that can be fused with the metacarpal base; it has been associated with carpal bossing and dorsal hand pain. The os hamuli forms when the hook of the hamate fails to fuse with the body of the hamate. The primary differential consideration is a chronic hook of hamate fracture; however, if the bone has smooth, well-corticated, and rounded margins it more likely represents an accessory ossicle. Other, ossicles, including the lunula, os triangulare, os trapezium secundarium, and os epilunate, are also occasionally observed but are less commonly clinically relevant.

In some individuals, additional accessory tendons and muscles may be present and should not be mistaken for pathology. For instance, the accessory abductor digiti minimi is the most common accessory muscle of the wrist with a prevalence of up to 24%. The muscle is radial and volar to the pisiform and inserts with the abductor digit minimi on the ulnar margin of the fifth metacarpal base. It can be readily identifiable as muscle tissue projecting into Guyon’s canal at the level of the pisiform because there normally should not be any muscle in Guyon’s canal at that level [42].

The extensor digitorum brevis manus is an accessory muscle that occurs in the fourth extensor compartment and can be unilateral or bilateral. The origin of the muscle can be variable, although it most commonly arises from the dorsal wrist capsule deep to the extensor retinaculum [42]. It usually inserts onto the extensor hood of the index or middle fingers but can occasionally insert onto the fourth and fifth fingers [43,44]. Clinically, it can be mistaken as a mass along the dorsal aspect of the wrist.

A bifid median nerve is a common anatomic variant with an incidence of approximately 2.8%. The median nerve can bifurcate into two nerve bundles in the distal forearm and appear as a bifid median nerve in the carpal tunnel. The bifid median nerve can also appear with an accessory artery, the persistent median artery, which lies between the two nerve bundles. Identifying this variation as well as the presence of the accessory artery can be of importance in avoiding injury to these structures during carpal tunnel release [45].

Occult fractures and their complications

MRI is the best imaging modality for evaluating radiographically occult wrist fractures, fracture complications, and osseous contusions (Figure 1). The scaphoid is important for carpal stability and acts as a stabiliser between the two carpal rows. Anatomic snuff box tenderness is a sensitive test for scaphoid fractures, but pain and tenderness over the scaphoid tubercle are more specific. Clinical examination alone is unable to assess for complications of scaphoid fractures.

Scaphoid fractures account for approximately 60-90% of all wrist fractures, and 12% of scaphoid fractures fail to heal and require surgical treatment [26,46]. Due to the unique blood supply of the scaphoid, fractures through the proximal pole are predisposed to delayed union, nonunion, and osteonecrosis. Radiography, including posteroanterior (PA), lateral, semipronated oblique and PA with ulnar deviation views, is the first-line imaging modality for detecting complications related to scaphoid fractures. However, MRI is more sensitive in detecting radiographically occult fracture lines and bone marrow oedema. If MRI demonstrates a fracture line with cyst formation on either side, there is suggestion of nonunion.

Scaphoid nonunion advanced collapse (SNAC) wrist occurs when persistent nonunion of the scaphoid results in the development of osteoarthritis and functional impairment about 5-10 years after injury. “Humpback deformity” is secondary to malunion of a scaphoid waist fracture with volar angulation of the proximal and distal poles. The angulated dorsal surface of the scaphoid forms the palpable “humpback” [47].

Treatment of scaphoid fracture complications varies depending on the ability to tolerate surgery and the viability of the bone. Patients who are unable to tolerate surgery for scaphoid nonunion may be candidates for non-operative management with casting for 4-6 weeks. In scaphoid nonunion with preserved vascularity, a non-vascularised bone graft fixated with screws is able to achieve successful union. If there is poor blood supply or osteonecrosis seen on MRI and/or absence of bleeding at surgery, vascularised bone grafting is performed. Scaphoid nonunion with osteoarthritis requires salvage procedures such as limited or total wrist arthrodesis, proximal row carpectomy, scaphoid excision, or arthroplasty.

MRI is also useful in detecting other, less frequent fractures involving the carpal bones. Hook of hamate fractures can occur by direct trauma or avulsive force from the transverse carpal ligament. These avulsion injuries are associated with racquet sports and golf [47]. Untreated fractures can progress to nonunion and may rarely require surgical excision for symptomatic relief [47].

Scaphoid

Preiser’s disease is the idiopathic form of osteonecrosis of the scaphoid bone, although most cases have a history of repetitive micro-injuries or overloading [11]. However, most cases of scaphoid osteonecrosis occur following proximal pole or waist fractures, and osteonecrosis has been reported in approximately 30% of scaphoid waist fractures and 80-100% when the fracture is located in the proximal pole [46]. Clinical examination cannot alone detect complications of scaphoid fractures. MRI is useful for determining the presence of osteonecrosis. If MRI demonstrates preserved normal fatty signal in the proximal fracture fragment, the finding strongly suggests viable bone marrow. However, mummified fat in early osteonecrosis may demonstrate the “normal” high signal intensity of fat on T1W images without fat saturation resulting in false negative findings. Otherwise, early osteonecrosis demonstrates low signal intensity of necrotic bone with respect to surrounding normal bones on T1W images and variable signal on fluid-sensitive, FS images. Contrast enhancement of the bone marrow suggests preserved vascularity. Homogeneous enhancement suggests viable bone marrow, while the absence of enhancement suggests osteonecrosis, and heterogeneous enhancement may represent the coexistence of osteonecrosis with variable viable tissue (Figure 5). However, fibrous tissue ingrowth can result in some enhancement and lead to false negative interpretation [48-50].

Lunate

Kienböck disease is osteonecrosis of either a part or all of the lunate. Osteonecrosis may result from repetitive microtrauma, interruption of blood supply including acute fracture, hypercoagulability, steroid use, altered biomechanics in association with negative ulnar variance, decreased radial inclination, and type I lunate morphology. A type II lunate with its extra articulation to the hamate may resist against fractures. Kienböck disease typically presents with dorsal-sided pain, restricted motion, weakness, and swelling. On clinical examination, there is focal tenderness between the extensor digitorum and extensor carpi radialis tendons with provoked pain on extension. Isolated lunate fractures are very rare, accounting for approximately 1-2% of wrist fractures [51]. The proximal pole of the lunate can be predisposed to ischaemia because it lacks soft tissue attachments. Palmar pole fractures can result in osteonecrosis due to interruption of the single arterial supply, which is observed in up to 20% of the population [26,31]. MRI findings of lunate osteonecrosis are those described previously when discussing scaphoid osteonecrosis (Figure 14). The Lichtman staging of Kienböck disease is important for patient management (Table 3).

Figure 14

Kienböck Disease. 44-year-old female with a few months of left wrist pain. Coronal T1W magnetic resonance image shows diffuse low T1 signal throughout the lunate (arrow), most notable at its radial aspect, compatible with osteonecrosis with partial collapse. Ulnar negative variance is also suggested on this image

Ulnolunate abutment and ulnar impingement

Ulnolunate abutment, also known as ulnocarpal loading or ulnar impaction syndrome, is characterised by ulnar wrist pain, swelling, and limited range of motion. The chronic impaction between the ulnar head and TFCC, lunate, and triquetrum can result in a spectrum of pathologic changes including degenerative tearing of the TFCC, lunate/triquetral chondromalacia, tearing of the LTL, or osteoarthritis of the ulnocarpal and DRUJ (Figure 15). These findings most commonly occur with ulnar positive variance but can occasionally occur with neutral or negative variance [52].

Figure 15

Ulnolunate abutment. 47-year-old male with bilateral wrist pain, left greater than right. Coronal T2W fat-suppressed magnetic resonance image shows bone marrow oedema in the lunate with subchondral cyst-like changes along the proximal ulnar aspect (arrow). There is significant central triangular fibrocartilage disc (TFC) thinning although no perforation was seen. These findings can be seen with ulnolunate abutment. The peripheral TFC appears normal. Note the intact meniscus homologue (arrowhead)

Ulnar impingement is a distinct entity from ulnolunate abutment. It is a syndrome characterised by a shortened distal ulna (negative ulnar variance) that impinges on the distal radius proximal to the sigmoid notch. The clinical findings are similar to those of ulnolunate abutment; however, there is pain on pronation and supination of the forearm and with compression of DRUJ. MRI findings include erosive changes at the level of the radius with osseous remodelling. MRI can be useful to detect earlier findings such as bone marrow oedema prior to the remodelling [52].

Triangular fibrocartilage complex

TFCC injuries most commonly occur from axial loading upon the extended wrist in pronation. Racquet sport athletes may present with TFCC injuries due to repetitive distraction loading on the volar aspect of the wrist, and ulnar positive variance can predispose to TFCC injuries. Patients with TFCC pathology present with ulnar-sided wrist pain, variable instability, and often a clicking or snapping sensation with rotation [26,32]. Due to the complex anatomy of the TFCC, high-resolution MRI with high SNR and high contrast resolution is required for accurate assessment. No specific imaging features are able to discriminate traumatic from degenerative tears.

TFCC injuries can be organised by Palmer’s Classification (Table 4) into either traumatic/acute (Class 1) or degenerative/chronic (Class 2). Class 2 tears are often the result of ulnolunate abutment from positive ulnar variance, rarely an elongated ulnar styloid process, or secondary to ulnar lengthening from radial fracture malunion. Class IA lesions appear as 1–2 mm sagittally-oriented tears ulnar to the radial aspect of TFC (Figure 16). These tears are in the avascular portion of TFC and are usually debrided due to poor healing [53]. Class IB lesions are traumatic avulsions from its attachment at the ulnar fovea proximal or distal laminae (Figure 17). These tears can be associated with fractures through the base of the ulnar styloid and injury to the palmar and dorsal radioulnar ligaments. The location of the tear is in the well-vascularised periphery of TFC and may heal. This is the most commonly missed tear. Excessive fluid accumulation and focal synovitis can be indirect signs of a tear. However, the intermediate to increased T2W signal of the ligamentum subcruentum can mimic a tear in this region [26,53]. Class IC lesions are traumatic avulsions from the peripheral volar attachments at the lunate or triquetral bones, specifically the ulnolunate and ulnotriquetral ligaments. If abnormal signal is seen within the ligaments on fluid-sensitive sequences, a tear is likely. The most advanced of these are Class ID lesions, which are traumatic avulsions from the radial attachment in the region of the sigmoid notch. They may be associated with distal radius fractures. These lesions are also usually debrided because the tissue is relatively avascular, which results in poor healing [26,53].

Table 4. Palmer’s classification of TFCC injuries

Figure 16

Central triangular fibrocartilage disc (TFC) tear. 26-year-old male with ulnar-sided right wrist pain after injury 9 months prior. Coronal T1W fat-suppressed magnetic resonance arthrography image after single compartment radiocarpal joint injection shows a full-thickness perforation through the central TFC disc (arrow). T1W hyperintense contrast is seen extending through the defect and into distal radioulnar joint, consistent with a Palmer 1A lesion

Figure 17

Peripheral triangular fibrocartilage complex (TFCC) tear. 49-yearold male with dorsal and ulnar-sided wrist pain following trauma. Coronal T2W fat-suppressed magnetic resonance image shows partial tearing of the triangular ligament of the peripheral TFCC at its ulnar styloid attachment (arrow) and a small distal radioulnar effusion with synovial proliferation, which commonly accompanies TFCC tears (arrowhead), consistent with a Palmer 1B lesion. Additional injuries to the distal radioulnar ligament and extensor carpi ulnaris tendon subsheath are not shown

Class IIA lesions represent degenerative wear and thinning of the articular disk without perforation. Focal abnormal signal intensity not extending to the surface of TFC probably represents mucoid degeneration. Class IIB lesions demonstrate progressive degeneration with findings of IIA lesions and additional chondromalacia of the lunate, triquetrum, or distal ulna without perforation. Class IIC lesions demonstrate even more progressive change than IIB lesions with a frank perforation of TFC, and Class IID lesions demonstrate LTL perforation as well as all of the findings of class IIC lesions. These lesions result in the LTL instability. Finally, Class IIE lesions are Class IID lesions with ulnocarpal and occasional distal radioulnar osteoarthritis. If the LTL is sufficient, they can be treated with debridement and pinning. However, if this ligament is completely absent, arthrodesis may be necessary [53].

Communicating tears are full thickness defects in which single injection MRA shows extension of contrast from the radiocarpal to the DRUJ (Figure 6 and 16). Non-communicating tears comprise partial-thickness defects. Degenerative tears and perforations are age-related and usually occur in the central disc. Radial/central communicating tears are often bilateral and can be asymptomatic [26,32,33].

Direct MRA has the highest accuracy in detecting TFC tears, especially for central and radial-sided (Palmer IA, ID, and II) tears. Moderate accuracy is present for peripheral tears at the ulnar attachment (Palmer IB, IC); however, MRA is still considered superior to PD/T2W FS images due to the presence of fibrovascular tissue [26].

Intrinsic ligaments

The SLL and the LTL are the most important ligaments that support the proximal carpal row. Furthermore, they are the most commonly injured ligaments, and their disruption can lead to altered biomechanics. The most frequent mechanism of injury is extension, ulnar deviation, and carpal supination. Repetitive stress to the SLL from crutch walking and ulnar impingement of the LTL can also result in injury [26,33,34].

Tears of the dorsal SLL band at its lunate attachment are usually symptomatic and traumatic rather than degenerative. Degenerative perforations of the central SLL and LTL increase with age and can be asymptomatic. Tears of the SLL result in rotatory subluxation of the scaphoid and dorsal intercalated segmental instability (DISI) with dorsal tilting of the lunate (Figure 18). An SLL angle > 80^o is diagnostic for DISI. This is best measured on true lateral wrist radiographs. Although it can be estimated on MRI, MRI measurements may be unreliable and highly dependent on patient positioning. Zanetti et al. found the mean measurement of the SLL angle on sagittal MRI to be closely correlated with measurements taken on lateral radiographs when the wrist was positioned in 15^o of radial deviation. However, when the wrist was placed in the neutral position the measurement could differ from the radiographic measurement by 14^o, and when the wrist was placed in 15^o of ulnar deviation, the measurement could differ by 32^o [54]. DISI can result in abnormal loading on the wrist and eventually result in scapholunate advanced collapse (SLAC) wrist, which is characterised by proximal migration of the capitate and osteoarthritis of the radiocarpal and intercarpal joints. The abnormal SLL may demonstrate fraying, irregular margins, increased signal on fluid-sensitive sequences, or fibre discontinuity. These findings may be present with or without widening of the SLL interval. Full-thickness tears will allow abnormal communication of contrast between the radiocarpal and midcarpal joints [26,33,34].

Figure 18

Scapholunate ligament (SLL) tear with dorsal intercalated segmental instability (DISI). 42-year-old male with left wrist pain after a fall. A) Coronal T2W fat-suppressed (FS) magnetic resonance (MR) image shows full-thickness tearing of the central band of the SLL (arrow) with widening of the scapholunate interval. B) Sagittal T2W FS MR image shows dorsal tilt of the lunate (arrow) compatible with DISI. C) Axial T2W FS MR image shows that the dorsal and volar bands of the SLL (arrow and arrowhead, respectively) are poorly defined and thickened, compatible with high-grade tearing

Disruption of the LTL results in volar intercalated segmental instability (VISI) with volar tilting of the lunate. This finding can be a normal variant in patients with joint laxity. The SLL angle < 30^o and capitolunate angle > 30^o are suggestive of VISI. Similar to the SLL, the dorsal band is best seen on axial imaging and the membranous component is best seen on coronal imaging [26,33,34]. The lunotriquetral articulation is usually obliquely oriented with respect to the short axis of the wrist. If there is clinical concern for LTL injury, an oblique axial sequence that is perpendicular to the LTL articulation may be helpful to improve characterisation of the dorsal and volar bands of the ligament.

Extrinsic ligaments

Tears of the volar extrinsic ligaments may contribute to DISI. The ulnar-sided volar ligaments contribute to the stability of the TFCC. Peripheral tears of the TFCC can result in injury to the volar ulnolunate and ulnotriquetral ligaments. Tears of the dorsal radiocarpal ligaments can lead to VISI, while a tear of the dorsal intercarpal ligament can lead to DISI.

Injuries of the extrinsic ligaments can be concurrently found with intrinsic ligament injury and carpal instability. Torn ligaments demonstrate discontinuity of fibres, while partial tears would show irregularity of the fibres and/or increased signal on fluid-sensitive sequences (Figure 19) [55].

Figure 19

Dorsal intercarpal extrinsic ligament sprain. 47-year-old female with right-side dorsal wrist pain after fall. Axial T2W fat-suppressed magnetic resonance image shows an oedematous and ill-defined dorsal intercarpal ligament especially along its triquetral attachment (arrow)

Flexor tendon injuries

Flexor tendon injuries in the hand can be classified into five anatomic zones (Figure 20). The wrist is composed of zone IV, the tendons within the carpal tunnel, and Zone V, the tendons extending from the myotendinous junction to the proximal aspect of the carpal tunnel. Zone IV injures are rare due to the protective nature of the transverse carpal ligament. However, a crush injury could result in damage to the carpal tunnel flexor tendons and the median nerve. Zone V injuries can result in damage to the ulnar and radial arteries, ulnar and median nerves, as well as multiple flexor tendons [56]. Flexor tendon tenosynovitis can occasionally be seen in the setting of inflammatory arthritis or in athletes with overuse injuries, such as those who play racquet sports or golf [26]. The section on the lesser fingers further discusses the various flexor tendon zones and related injuries in greater detail.

Figure 20

Illustrated schematic of the flexor and extensor tendon zone injuries

Extensor tendon injuries

The most common form of tenosynovitis is known as DeQuervain’s tenosynovitis and involves the first extensor compartment tendons, including the abductor pollicis longus and extensor pollicis brevis tendons (Figure 21). DeQuervain’s tenosynovitis typically presents in middle-aged women bilaterally, young mothers carrying babies, and racquet sport athletes. Patients present with pain and swelling over the radial styloid process with increased pain during thumb extension and abduction [26,57].

Figure 21

DeQuervain’s tenosynovitis. 37-year-old female with radial-sided wrist pain and clicking. Axial T2W fat-suppressed magnetic resonance image shows thickening and signal heterogeneity of the first extensor compartment tendons consistent with tendinosis and distension of the tendon sheath (arrow) compatible with DeQuervain’s tenosynovitis

Proximal intersection syndrome is a usually chronic, overuse, inflammatory condition affecting the dorsal radial aspect of the distal forearm 4–5 cm proximal to the radiocarpal joint, where the first and second extensor compartment tendons cross each other. This syndrome results from repetitive friction that can lead to tenosynovitis, stenosing tenosynovitis, and occasionally adventitial bursitis. The condition is associated with rowing and racquet sports and is less commonly from direct trauma [26,57].

The ECU tendon can present with tenosynovitis, tendinopathy, subluxation, or dislocation due to tendon sheath disruption and tendon rupture (Figure 22). Although it can be seen in normal individuals, ECU tendon subluxation can also be related to an acute injury, and the patient history and physical examination may be needed to diagnose pathologic subluxation [26,57].

Figure 22

Extensor carpi ulnaris (ECU) interstitial tear. 22-year-old male professional baseball player with left wrist pain and instability after injury 12 weeks prior. A) Axial T2W fat-suppressed (FS) magnetic resonance (MR) image of the wrist at the level of the ulnar groove with the wrist in neutral position shows increased signal in the ECU tendon with a small tenosynovial effusion (arrow). B) Axial T2W FS MR image of the wrist at the level of the ulnar groove with the wrist in supination shows an abnormal boomerang shaped morphology of the ECU tendon, which is draped around the ulnar margin of the groove (arrow). At surgery there was an interstitial tendon ear with insufficiency of the overlying retinaculum that allowed pathologic tendon subluxation

Rheumatoid arthritis (RA) frequently presents with tenosynovitis and tendinopathy because the wrist tendons are surrounded by a synovial sheath or bursa. However, they rarely present with tendon tears. Approximately 50–64% of RA patients demonstrate extensor tendon involvement, with the ECU being the most commonly affected tendon in patients with early RA. On MRI, tendinopathy is characterised by heterogeneous high signal intensity and thickening on fluid-sensitive sequences, and active tenosynovitis will demonstrate tendon sheath effusions with enhancement [57].

Distal intersection syndrome, also known as decussation syndrome, is caused by the third extensor compartment tendon crossing the second compartment tendons just distal to Lister’s tubercle, which arises from the dorsum of the distal radius (Figure 23). The second and third extensor compartment tendon sheaths communicate via a foramen, which can allow the spread of inflammation between the two. Patients present with pain and swelling over Lister’s tubercle and occasionally focal crepitus with movement of the thumb. The condition is most commonly post-traumatic but can also be seen with various rheumatologic and degenerative diseases [26,57].

Figure 23

Distal Intersection (Decussation) Syndrome. 69-year-old female with dorsal wrist pain. Axial T2W fat-suppressed magnetic resonance image demonstrates mild peritendinous oedema of the extensor pollicis longus tendon and mild tenosynovitis (third compartment) (arrow) as it crosses over the second extensor tendons at the level of the carpus

Entrapment neuropathies

Entrapment neuropathies are peripheral neuropathies characterised by pain and/or loss of function secondary to nerve compression.

Carpal tunnel syndrome is the most common nerve entrapment in the upper extremity. It can be idiopathic or related to various causes of nerve compression, including lunate dislocation, haematoma, or flexor tendinopathy/tenosynovitis related to rheumatologic disorders. Additional aetiologies include diabetes, hypothyroidism, pregnancy, obesity, amyloid deposition, osteoarthritis of the first carpometacarpal (CMC) joint, or space-occupying lesions along the nerve. Clinical symptoms include pain, tingling, and numbness in the thumb, and index and middle fingers. Physical exam findings include atrophy and weakness of the thenar musculature with impaired sensation extending from the thumb to the lateral fourth fingers and with preserved sensation over the thenar eminence. A positive Tinel test, or tingling sensation in the distribution of the nerve elicited by light percussion over the nerve, is an additional sign that supports diagnosis of the disease. MRI findings demonstrate increased signal within the median nerve, enlargement of the nerve at the level of the pisiform, and flattening of the nerve at the level of the hook of hamate (Figure 24). Cross-sectional area (CSA) measurement of the median nerve, including absolute measurements at various points within, proximal and distal to the carpal tunnel, and ratio measurements of CSA in the distal forearm and within the carpal tunnel, can be sensitive in detecting cases of carpal tunnel syndrome. On ultrasound normal median nerve CSA has been reported between 9 and 11 mm². Although ultrasound measurements have been well-assessed in the literature, MRI measurements are less established. MRI measurements of the median nerve CSA are reportedly routinely larger than those on ultrasound and similar to those measured on cross-sectional cadaveric specimens [58]. A recent work by Ng et al. has suggested a CSA > 15 mm² either proximal or distal to the carpal tunnel as a cut-off for detecting carpal tunnel syndrome, and CSA > 19 mm² as a cut-off for severe carpal tunnel syndrome [59]. Additionally, on axial imaging of the wrist a ratio measurement between the long axis of the median nerve seen in cross section and its short axis at the carpal tunnel inlet of greater than 3:1 may also help to detect cases of carpal tunnel syndrome suggested on nerve conduction testing [60]. Other secondary MRI findings include thickening and bowing of the flexor retinaculum, thenar muscle oedema, and/or atrophy and reactive hypertrophy of the lumbrical muscles. Post-contrast imaging shows enhancement of oedematous nerves and reduced enhancement in ischaemic nerves [26,61].

Figure 24

Median nerve compression due to flexor retinacular thickening. 60-year-old female with chronic numbness in her hand. Sagittal T2W fat- suppressed magnetic resonance image shows flattening and compression of the median nerve (arrow) with thickening of the overlying distal flexor retinaculum, and increased signal in the nerve proximally (arrowhead)

Complications related to carpal tunnel release are estimated at between 3 and 19%, and surgical re-exploration is necessary in up to 12% of cases [61]. Approximately 1–25% of patients have recurrence of symptoms after surgery [61]. Common causes of recurrent carpal tunnel syndrome include incomplete resection/regrowth of the flexor retinaculum, flexor tenosynovitis, fibrous proliferation, iatrogenic median nerve damage, and extrinsic compression, which can be caused by ganglion cysts or accessory muscle bellies. Regrowth of the flexor retinaculum appears on MRI as a well-defined, continuous linear area of low signal intensity superficial to the nerve and thickening of the retinaculum deep to the subcutaneous postoperative scaring. Fibrosis can present as low T1 signal with ill-defined margins within the nerve [61].

Wartenburg syndrome, also known as cheiralgia paresthetica, is a rare syndrome related to isolated pathology of the superficial radial nerve resulting from nerve entrapment between the brachioradialis and extensor carpi radialis longus tendons, which is often due to compression from a watch strap or bracelet. Clinical symptoms include pain over the dorsoradial aspect of the hand [62].

Ulnar nerve compression can occur proximal to Guyon’s canal (Type I) or within the canal (Type II) resulting from mass effect related to ganglion cysts, neuromas, or accessory muscles. On rare occasions, ulnar artery pseudoaneurysm, hypertrophy of the flexor carpi ulnaris tendon, osteoarthritis of the pisotriquetral joint, or hamate/pisiform fractures can lead to compression. Clinically, patients present with fourth and fifth finger sensory loss and weakness. Physical examination demonstrates sensory deficits of the little finger and the ulnar half of the ring finger, as well as a “claw” deformity of the ring and little fingers secondary to intrinsic muscle weakness. MRI findings of Guyon’s canal syndrome demonstrate a space-occupying lesion along the course of the nerve with an oedematous ulnar nerve proximal to the canal, flattening of the nerve within the canal, and increased signal intensity of the nerve on fluid-sensitive sequences (Figure 2). Secondary signs include muscle oedema and enhancement of the hypothenar muscles, third and fourth lumbricals, and interosseous muscles related to denervation [26,62].

A rare cause of ulnar-sided pain is hypothenar hammer syndrome, which is secondary to thrombosis of the ulnar artery overlying the hamate bone as it exits Guyon’s canal. The name of the syndrome comes from patients repetitively hammering objects with the hypothenar aspect of their palms, which causes trauma to the underlying ulnar artery. This results in intimal and medial wall degeneration and thrombosis with distal emboli that can occlude the distal digital arteries and symptomatic ischaemia [63]. MRI can demonstrate an abnormal flow void of the ulnar artery to help suggest this diagnosis (Figure 25).

Figure 25

Hypothenar hammer syndrome. 34-year-old male with left ring and middle finger pain beginning a few weeks after doing mechanical work on his car. A) Axial T2W fat-suppressed magnetic resonance image shows increased signal and surrounding soft tissue oedema along the ulnar third interdigital neurovascular bundle (arrow). B) Ultrasound colour Doppler short-axis image shows thrombosis of the ulnar artery more proximally at the level of the wrist (arrow)

Infection

Closed space infection of the flexor tendon sheaths can result in pyogenic flexor tenosynovitis. Increased pressure within the tendon sheath can lead to tendon rupture or necrosis. Alternatively, adhesions can lead to stenosing tenosynovitis with severe restriction of motion. Infection from the thumb and little finger tendon sheaths can spread into the radial and ulnar bursae and communicate with each other, resulting in a horseshoe abscess. Infection can also spread between the radial and ulnar bursae and rupture into the space of Parona, a potential space between the pronator quadratus fascia and FDP tendon sheath. This is significant because it can allow the spread of infection proximally into the forearm [36].

Bones and joints

The bones and joints are typically evaluated for contusion, fracture, malalignment, dislocation, or joint effusion in the setting of injury. For fractures involving the articular surface, the amount of involvement should be quantified because it can be an indicator of joint stability. Any fracture displacement should also be described, including the size of any bony fragments. The joints are also evaluated for cartilage integrity and for any evidence of a degenerative or inflammatory arthropathy. Radiographs are typically useful for aiding in evaluation. Additionally, the soft tissues of the lesser digits are evaluated for the presence of haematoma, fluid collection, or oedema in the setting of injury.

Ligaments and volar plate

On MRI, oedema surrounding the ligaments and volar plates typically indicates acute injury of that structure. Additionally, the structures are evaluated for the presence of partial or full-thickness tearing. The location of any tears should be described, along with any osseous/periosteal involvement. Ligamentous injuries most commonly involve the PIP joint, and dorsal dislocations can result in volar plate tears. Any imaging evidence of ligamentous/volar plate malpositioning or trapping should be described in the report to aid clinical management.

Extensor mechanism

Extensor mechanism injuries are commonly classified according to anatomic location. The Verdan system is most commonly used, which includes eight zones (Figure 20) [66]. The odd-numbered zones are located at the joint levels. Injury location, percentage of tendon involvement and any adjacent osseous or soft tissue involvement should be described with detail in the report.

Zone 1 injuries are the most common and are usually closed. They occur at the level of the terminal tendon and frequently arise when the finger is extended and there is forceful flexion of the DIP joint resulting in an avulsion injury, known as a mallet finger. Osseous avulsion from the dorsal base of the distal phalanx can occur. Zone 1 injuries are typically treated by splinting the finger with the DIP joint in extension.

Zone 2 injuries are usually due to a laceration and occur at the level of the middle phalanx. These injuries commonly involve the conjoined tendon(s) and/or triangular ligament. Injury to one of the conjoined tendons is usually managed conservatively. Surgical repair is usually indicated if the injury involves the triangular ligament and/or both tendons.

Zone 3 injuries can be open or closed and occur at the PIP joint level. Closed injuries are usually caused by a blow to the dorsum of the finger, forceful flexion of the PIP joint, or volar joint dislocation. These injuries can involve the distal central tendon slip with possible osseous avulsion of the dorsal middle phalangeal base, but they can also involve the lateral slips. Many injuries can be treated conservatively by splinting with the PIP joint in extension. However, open injuries, displaced osseous avulsion fragments, and instability may necessitate surgical intervention.

Zone 4 injuries at the level of the proximal phalanx often result from lacerations and can involve the central slip, lateral slips, medial and lateral lumbrical bands, and extensor hood. Most injuries in this zone are partial tears without significant retraction since the extensor mechanism in this region is broad. Injuries in this zone are usually treated conservatively with splinting with the PIP joint in extension. Higher grade injuries usually require surgical intervention.

Zone 5 injuries at the level of the MCP joint commonly occur at the index finger MCP joint with flexion. These injuries are usually due to a biting mechanism and involve the extensor tendon with either a complete or partial tear. The sagittal band and joint capsule can also be involved (Figure 29). Oblique injuries involving the sagittal band can be open but are more commonly closed; and can involve subluxation or dislocation of the extensor tendon, often resulting in a snapping sensation for the patient. There is concern for infection with open injuries, including septic arthritis if there is involvement of the joint capsule. Surgical repair is frequently required for zone 5 injuries.

Figure 29

Focal extensor digitorum communis (EDC) tendon tear at extensor tendon zone 5 with tearing of the ulnar sagittal band. Axial T2W fat-suppressed magnetic resonance image of a 37-year-old female with pain and swelling dorsal to the left third metacarpophalangeal joint shows partial tearing of the EDC tendon with tendon thickening consistent with tendinopathy. This is associated with a focal tear of the ulnar sagittal band as it attaches onto the tendon (arrow)

Zone 6 injuries in the dorsum of the hand are usually due to laceration. The extensor tendons are highly superficial in this region. Multiple extensor tendons can be involved with a single injury. Clinical exam may not demonstrate a significant loss of extension with single tendon or partial tendon lacerations due to connections between the tendons. Surgical repair is required.

Flexor mechanism

MRI is useful for evaluating flexor tendon injuries. T2W sequences typically demonstrate fluid signal in the region of a recent tear [27]. Important imaging findings include whether the tear is partial-thickness (with an estimate of the cross-sectional percentage of tendon involvement) or full-thickness. Location of the tear and any tendinous retraction should be clearly described, in addition to the presence of any osseous avulsions and any displacement of the bony fragment.

Flexor tendon injuries have been classified into five anatomic zones by Kleinert et al. and Verdan (Figure 20) [67,68]. Flexor tendon injuries are more commonly open, and often due to laceration. Less commonly, they may be closed and related to avulsion. The most common type of closed avulsion injury is a tear of the distal FDP tendon. This usually occurs due to forced hyperextension of the DIP joint during active flexion, in activities such as football or rugby, and most often involves the ring finger (Jersey finger). Leddy and Packer initially classified these injuries into three predominant types [69], with additional types subsequently described. Type I injuries result when the tendon is retracted into the palm. Type II injuries occur when the tendon is retracted to the PIP joint level. Type III injuries are present when there is an avulsed bone fragment, with the fragment held in place by the A4 pulley; type IV injuries occur when the bony fragment is present, but the tendon has been avulsed from the fragment [64]. The majority of these injuries require surgical repair, and type I injuries are often urgent due to blood supply compromise [27]. Isolated FDS avulsions are rare, and FDS injuries usually occur in conjunction with FDP injuries.

Flexor tendon laceration commonly occurs in the mid substance rather than at the insertion [70] and is classified according to its location in the zones described by Kleinert et al. and Verdan (Figure 30). Injuries in zones 2 through 5 can involve the neurovascular bundle and require emergency surgery [27]. Zone 2 injuries are the most common and are associated with the highest rate of complications. Although traditionally zone 2 injuries were not repaired surgically due to the high rate of complications, these injuries are currently repaired surgically due to advances in understanding of these injuries and surgical technique. Surgical repair for these injuries ideally is performed within 24 hours in current practice [27].

Figure 30

Distal flexor digitorum profundus (FDP) tendon laceration in flexor zone 1. Sagittal T2W magnetic resonance fat-suppressed image of the left middle finger in a 23-year-old male who suffered a laceration demonstrates the complete FDP tendon tear distal to the flexor digitorum superficialis insertion (arrow) with a 1.1 cm tendon gap

Injuries to the annular pulleys are best visualised on axial imaging (Figure 31). Rock climbing is a common cause of pulley injury, most frequently involving the A2 pulley of the ring finger [27]. Tears can either be partial or complete and can involve either a single pulley or multiple pulleys. A consequence of pulley tearing is bowstringing of the flexor tendon, which is variable depending on the number of pulleys torn and the positioning of the finger [27]. Comparison with the adjacent finger flexor mechanism can be helpful when trying to assess for subtle bowstringing on MRI. Fluid signal around the pulleys is usually seen in the setting of acute injury. Klauser et al. examined the degree of displacement of the flexor tendon from the volar surface of the proximal phalanx (the tendon-to-phalanx [TP] distance) at rest and with forced finger flexion on both MRI and ultrasound in elite rock climbers. They observed that a TP distance of more than 1.0 mm was a sign of pulley insufficiency and at least partial tearing and an increase in palmar tendon displacement of at least 1.0 mm with forced finger flexion compared with rest was also a sign of at least partial tearing. With forced finger flexion, a TP distance of more than 2.5 mm may be helpful in diagnosing a complete tear of either the A2 or A4 pulleys [71].

Figure 31

Low-grade A1 flexor tendon pulley injury. Axial T2W fat-suppressed magnetic resonance image of the third and fourth metacarpal heads in a 59-year-old female who sustained a low energy trauma to the middle and ring fingers shows mild thickening, intermediate signal, and surrounding oedema of the ring finger A1 pulley (arrowheads). There is mild oedema around the middle finger A1 pulley. However, the pulley fibres appear intact, uniform in thickness, and low in signal (arrow)

First CMC joint

Acute ligamentous injuries of the thumb can occur but are much less common than at the MCP joint. The most common internal derangement in this location is osteoarthritis. This is generally due to ligamentous laxity related to repetitive movements such as pinching or a single traumatic event. There is a higher incidence of first CMC osteoarthritis in women, which has been associated with hormonal influences. Radiography is the primary tool for assessing osteoarthritis, but MRI can be useful to look for ligament injuries, such as to the AOL, which can result in joint instability. Furthermore, MRI often detects signs of osteoarthritis earlier than radiography. Finally, MRI can be used to look for additional causes of radial-sided pain, such as DeQuervain’s tenosynovitis or radiographically occult fractures. Typical MRI findings include synovial proliferation and effusion, cartilage loss, bone marrow oedema with subchondral cyst-like changes, and marginal osteophytes. The stabilising ligaments may be thickened and intermediate in signal, indicating chronic scarring and degeneration. As with acute ligament injuries elsewhere, MRI findings include thickening and intermediate/increased signal within the ligament with periligamentous oedema, and a fluid signal intensity gap either within the ligament or at its osseous attachments, which indicates a partial or complete tear [76].

First MCP joint

UCL injuries are commonly related to hyperabduction of the thumb, particularly while grabbing an object such as a ski pole during a fall. As a result, this condition is commonly called skier’s thumb. The term gamekeeper thumb has been used synonymously, although it more commonly refers to chronic injuries, while skier’s thumb is generally reserved for more acute injuries. UCL tears most often occur distally at the proximal phalangeal attachment and may be associated with osseous avulsions; however, they can be proximal, near the metacarpal head attachment, or in the mid substance [77]. A Stener lesion occurs when the distal UCL tears and the overlying adductor aponeurosis becomes interposed between the UCL stump and the proximal phalangeal base. This prevents healing of the UCL and leads to joint instability and osteoarthritis [13].

MRI is very helpful in diagnosing and characterising UCL injuries. It has been reported that the sensitivity for complete proper UCL tears is 96%, while the specificity is 95% [78]. Injuries of the UCL can be divided into those that are acute or those that are chronic, and the MRI findings will differ. In acute UCL injuries there is commonly periligamentous oedema seen on fluid-sensitive sequences. The ligament may be thickened, ill-defined, and contain internal intermediate signal. A complete tear is diagnosed when there is a fluid signal gap within the ligament or at the ligament attachment on to the bone, and there may be an associated avulsion fracture with variable degrees of bone marrow oedema (Figure 36) [13]. If the avulsed osseous fragment is displaced by more than 5 mm, or if it involves more than 25% of the articular surface, surgery is indicated.

Figure 36

First metacarpophalangeal (MCP) ulnar collateral ligament (UCL) osseous avulsion in a 26-year-old male professional baseball player who injured his thumb while sliding into a base. Coronal T2W fat-suppressed magnetic resonance image of the thumb at the MCP joint depicts an osseous avulsion of the UCL from the proximal phalangeal base (arrowhead) with subtle marrow oedema in the proximal phalangeal base and a fluid gap between the torn ligament and the proximal phalanx (shorter arrow). However, no Stener lesion is seen and the adductor aponeurosis is noted overlying the torn UCL (longer arrow)

In chronic injuries, the soft tissue and marrow oedema usually resolve, and the ligament may appear thickened and irregular, which reflects chronic scarring. There may be a gap between the bone and the ligament stump, and the ligament stump may be retracted and globular. In these cases, the adductor aponeurosis can lie deep to the UCL stump, which forms the classic “yo-yo on a string” appearance described in the Stener lesion (Figure 37). In this finding, the string represents the aponeurosis, while the yo-yo is the thickened ligament stump [79]. Some authors have noted that an intact accessory band or sesamoid-metacarpal ligament may prevent the development of a Stener lesion [80]. MRI may be helpful to assess the integrity of these structures, to determine whether a patient may be prone to developing a Stener lesion [64,74].

Figure 37

First metacarpophalangeal (MCP) ulnar collateral ligament osseous avulsion with Stener lesion in a 39-year-old female with left thumb valgus injury. A) Coronal T1W fat-suppressed (FS) gradient echo magnetic resonance (MR) image of the MCP joint shows a fracture fragment off the ulnar aspect of the proximal phalangeal base (arrow) that is displaced by 6 mm. This is an indication for surgical repair. B) Coronal T2W FS MR image of the same area shows a Stener lesion with a “yo-yo on the string” appearance. The arrowhead indicates the adductor aponeurosis (string), while the arrow shows the retracted ligament and osseous fragment (yo-yo)

RCL injuries are much less common than UCL injuries. Although most injuries are lower grade sprains and can be managed with immobilisation, higher grade partial and complete tears have been shown to contribute to joint instability, chronic ulnar deviation, and MCP osteoarthritis. As a result, these injuries are commonly treated surgically [79]. There are rare reports of a Stener-like lesion involving interposition of the abductor aponeurosis and the RCL, which also requires surgical intervention [81].

Trigger finger/trigger thumb

Trigger finger/trigger thumb results in a painful click with restricted range of motion. This can produce a locked digit that must be manually unlocked. It usually occurs in the fifth and sixth decades of life and is more common in diabetic patients. It is caused by flexor tendon and pulley fibrocartilaginous metaplasia, usually the proximal margin of the A1 pulley. MRI may demonstrate tendon thickening and intermediate intrasubstance signal, indicating tendinosis, and there may be thickening of the pulley (Figure 38). When this occurs in the thumb, it may spontaneously resolve. However, surgery is indicated for persistent triggering.

Figure 38

Trigger thumb. 65-year-old male who was experiencing thumb clicking and limited range of motion. Axial T2W fat-suppressed magnetic resonance image of the thumb at the first metacarpophalangeal level shows mild flexor pollicis longus tendinosis with thickening and intermediate intrasubstance signal (arrow). Additionally, the A1 pulley is mildly thickened and intermediate in signal (arrowhead)